Luc Montagnier Quotes

First of all, many human diseases are influenced by, if not caused by mutations in genes.

Some genetic variants can be informative about one's risk for Parkinson's disease and Alzheimer's disease.

Genes work with probabilities; they don't work with certainties. So most things that you're looking at with these genetic tests, it's not like you're condemned to automatically get the disease or the syndrome. There's a lot of factors in play there.

[H]as the CDC ever published a story about the dangers of catching diseases when you sleep with illegal aliens?

It is often seen that in households where all members are exposed to the same danger, or again in schools or troops where everyone lives the same life, disease does not strike everyone indifferently.

In the past, geneticists have looked at so-called disease genes, but a lot of people have changes in their genes and don't get these diseases. There have to be other parts of physiology and genetics that compensate.

The belief is growing on me that the disease is communicated by the bite of the mosquito... She always injects a small quantity of fluid with her bite - what if the parasites get into the system in this manner.

Many people assume the diseases that kill us are pre-programmed into our genes. High blood pressure by 55, heart attacks at 60, maybe cancer at 70, and so on... But for most of the leading causes of death, our genes usually account for only 10-20 per cent of risk.

Studies of immigration show that this resistance to heart disease is not just something in African or Chinese genes. When people move from low-risk to high-risk areas, disease rates skyrocket as they adopt Western diets and lifestyles.

human genetics has taught us that when it comes to our telomerase, we humans live on a knife edge. OK, simply put, yes, nudging up telomerase does decrease the risks of some diseases, but it also increases the risks of certain and rather nasty cancers.

We and other groups are seeing clear statistical links between telomere shortness and risk for a variety of diseases that are becoming very common, such as cardiovascular disease, diabetes and certain cancers.

Even if it had not been possible to reproduce the disease in animals and consequently to verify the hypothesis, this simple observation would have been sufficient to demonstrate the way in which the disease was propagated.

I don't like rats, but there's not much else I don't like. The problem with rats is they have no fear of human beings, they're loaded with foul diseases, they would run the place given half the chance, and I've had them leap out of a lavatory while I've been sitting on it.

Your age is your No. 1 risk factor for almost every disease, but it's not a disease itself.

Genes can't possibly explain all of what makes us what we are.

A carrier with a venereal disease can have many partners, but only those whose mental and physical immune systems are weak will be susceptible to it.

We need 10,000 genomes, not 100, to start to understand the link between genetics, disease and wellness.

The fact that I was fortunate enough to escape contagion, in spite of frequent, sometimes daily contacts with the disease, was because I soon guessed how it spread.

One characteristic aspect of ageing is the increased susceptibility to disease, particularly age-related diseases such as cardiovascular diseases and cancer.

One of the things about genetics that has become clearer as we've done genomes - as we've worked our way through the evolutionary tree, including humans - is that we're probably much more genetic animals than we want to confess we are.

You don't have free will when you have lung cancer.

Left to their own devices, epidemic diseases tend to follow the same basic process: A virus or bacteria infects a host, who typically becomes sick and in many cases dies. Along the way, the host infects others.

In today's world, new infections and diseases can spread across the country and even across the world in a matter of days, or even hours, making early detection critical.

You can have an epidemic in a state. You can have it in a region. You can have it in a country where the critical level of disease passes a certain threshold, and we call that an 'epidemic threshold.'

But with each passing year and each new study, the evidence for the genetic contribution to individual and group differences becomes more firmly established than ever.